The drug co-trimoxazole is known by dozens of brand names, including Bactrim or Septra. Co-trimoxazole is a sulfa drug, a remnant of the era before penicillin and the other antibiotics. Sulfa drugs do not target invading microbes as narrowly as the antibiotics and so have become notorious for their side effects. According to the Physician's Desk Reference, Septra can cause "nausea, vomiting, anorexia," and "bone marrow depression," and also includes "rash, fever, [and] leukopenia" among its side effects. Even the drug's manufacturer, Burroughs Wellcome, strongly recommends against using Septra for more than two weeks, in children or adults.
Source: WITH THERAPIES LIKE THIS, WHO NEEDS DISEASE?

Treatment with combined trimethoprim/sulphamethoxazole (=co-trimoxazole) is especially serious for the functioning and fine structure of mitochondria in nucleated (eukaryotic) unicellular and multicellular species (protozoa, fungi, plants, animals, humans). Mitochondria - so-called organelles - are the major suppliers of energy in human cells (except in red blood cells). They are endosymbionts (former bacteria with a double membrane). They contain remnants of their ancestral genome. This mitochondrial DNA (mtDNA) is irreplaceable in the synthesis of protein sub-components of the respiratory chain. For respiration, activated electrons in the respiratory chain from nutrients using oxygen are built into the universal energy source for the entire cell, adenosine triphosphate (ATP). If the synthesis of precursors of DNA is harmed through chronic or high dose treatment with co-trimoxazole the mitochondrial DNA is damaged and altered which in consequence impairs mitochondrial proteins, as well as the proteins of the respiratory chain, and ATP production therefore decreases. This leads to increased oxidative stress and to an increase in toxic oxygen free radicals. A vicious circle is set up once the ATP levels reach a critical low, and if the special molecules which normally remove harmful oxygen intermediaries are all used up, then further DNA damage arises. The cell initiates programmed cell death, because the ion pumps which regulate the balance of the flow of manifold molecules of building supplies and working materials into and out of the cell necessary to maintain cell function, fail for lack of fuel in the form of ATP.
[...]
The above basic facts of cell biology apply, of course, with greatest force in rapidly maturing cells with short half-lives, especially the thymus-matured lymphocytes (T-cells), whose job is not only to recognise and, with the help of other immune cells eliminate, foreign proteins, but also to remove altered selfproteins without causing inflammation. If this cannot be done adequately because of infectious, toxic, nutritional, psychological or other overload, the body enters a state of emergency: the B-cell system is stimulated to produce antibodies and autoantibodies as well as macrophages and many inflammatory mediators and the entire metabolism is transformed. Over the short term, the body can deal with such a state of emergency. If this state persists, however, a chronic maturation deficit of T-lymphocytes (T-helper cell deficiency) arises, and the now permanently changed environment becomes the feeding ground (substrate) for the recycling activity of fungal parasites (in Greek, parasite means unwelcome scrounger) and as a consequence of B-cell activation, specific autoantibody profiles make the "HIV antibody test" turn positive*, just as in some autoimmune diseases such as rheumatoid arthritis and lupus erythematosus.

Under these conditions of highly acute state of emergency such as is found in "immune-suppressed patients," it does not require the wisdom of Solomon to see that the treatment induces precisely that which they seek to avoid, namely, an Acquired Immune Deficiency Syndrome (AIDS) induced through wrong medical practice.
Source: ACQUIRED IATROGENIC DEATH SYNDROME (AIDS)

* The significance of this is suggested in The Clap Doctor an article about Dr. Joseph Sonnabend: "Sonnabend discovered that as far back as 1969, doctors were treating PCP with sulfa drugs. In 1977, Dr. Walter Hughes of Tennessee had published an article in the NEJM - four years before Gottlieb noted his mysterious cases of PCP in another NEJM piece. Hughes had shown that in a placebo-controlled, double-blind trial, a drug called Bactrim prevented Pneumocystis in patients with compromised immune systems. This was an amazing discovery. PCP was a major killer. Now Sonnabend had a treatment. He immediately began to correspond with Hughes. As a result, he started to prescribe Bactrim and a similar drug, Septra, to all his patients with AIDS. That didn't prevent them from coming down with opportunistic infections, but it did save them from the deadly PCP. Sonnabend also called other community doctors with the news. They too began prescribing Bactrim and Septra."

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